Author

M. Zeng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Carr· 2 shared C. Liu· 2 shared D. Kong· 2 shared J. Chen· 2 shared X. Wang· 2 shared Z. Tang· 2 shared A. Baako· 1 shared A. Lipschutz· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

A. Carr

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Early life high fructose exposure disrupts microglia function and impedes neurodevelopment

Z. Wang, A. Lipschutz, Z.-L. Liu, A. Trzeciak, I. Miranda, C. Martinez de la Torre, T. Schild, T. Lazarov, W. S. Rojas, P. Saavedra, J. Romero-Pichardo, A. Baako, F. Geissmann, G. Faraco, L. Gan, , J. Perry

This study shows that early life high fructose exposure impairs microglia function and neurodevelopment, with GLUT5 deletion reversing these effects.

bioRxiv · Aug 14, 2023 · PreprintImmunology

Mechanistic basis for targeting homologous recombination defective liver cancer via synthetic lethality

M. Zeng, Z. Tang, L. Ren, H. Wang, X. Wang, W. Zhu, X. Mao, Z. Li, X. Mo, J. Chen, J. Han, D. Kong, J. Ji, A. Carr, C. Liu

This study identifies a novel subtype of HRD in HBVHCC caused by CDW19S perturbation and shows it can be targeted therapeutically.

bioRxiv · Jul 21, 2022 · PreprintCancer Biology

Synthetic lethality between TP53 and ENDOD1

Z. Tang, M. Zeng, X. Wang, C. Gao, P. Yue, X. Zang, H. Lou, J. Chen, D. Mu, D. Kong, A. Carr, C. Liu

This study identifies synthetic lethality between ENDOD1 and TP53, showing that their combined loss causes DNA damage and cell death, suggesting ENDOD1 as a cancer therapeutic target.

bioRxiv · Oct 14, 2020Cancer Biology