Author

J. Streeter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Research Focus:metabolism & bioenergetics· 2 Assay:bulk RNA-seq· 1 Organism:Drosophila· 1

Frequent Co-Authors

E. D. Abel· 4 shared L.-S. Song· 3 shared R. O. Pereira· 3 shared A. Hinton· 2 shared A. Marshall· 2 shared B. Glancy· 2 shared D. Hall· 2 shared J. Shao· 2 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersBiophysics

E. D. Abel

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cardiac MAO-A inhibition protects against catecholamine-induced ventricular arrhythmias via enhanced diastolic calcium control

Q. Shi, H. Malik, J. Streeter, J. Wang, R. Huo, R. M. Crawford, J. C. Shih, B. Chen, D. Hall, E. D. Abel, L.-S. Song, E. J. Anderson

This study shows that inhibiting MAO-A in cardiomyocytes reduces arrhythmias by improving diastolic calcium control following catecholamine stress.

bioRxiv · Sep 29, 2022Physiology

ATF4 Dependent Increase in Mitochondrial-Endoplasmic Reticulum Tethering Following OPA1 Deletion in Skeletal Muscle

A. Hinton, P. Katti, M. Mungai, D. Hall, O. Koval, J. Shao, Z. Vue, E. G. Lopez, R. Rostami, K. Neikirk, J. Ponce, J. Streeter, B. Schickling, S. Bacevac, C. Grueter, , E. D. Abel

This study shows that OPA1 deficiency in skeletal muscle increases MERC formation via ATF4, affecting calcium exchange and mitochondrial-ER tethering.

bioRxiv · Sep 12, 2022Biophysics

Practices for Measuring 3D Organelle Morphology and Generating Surfaces with Amira

E. Garza Lopez, Z. Vue, P. Katti, K. Neikirk, M. Biete, J. Lam, H. Beasley, A. Marshall, T. Rodman, T. A. Christensen, J. L. Salisbury, L. Vang, M. Mungai, S. AshShareef, S. Murray, , A. Hinton

The paper introduces a standardized protocol using Amira software to quantify 3D organelle morphology from SBF-SEM data, demonstrating its application to mitochondria and ER structures.

bioRxiv · Sep 25, 2021Biophysics

Cardiac Specific Overexpression of Transcription Factor EB (TFEB) in Normal Hearts Induces Pathologic Cardiac Hypertrophy and Lethal Cardiomyopathy

H. C. Kenny, E. T. Weatherford, G. V. Collins, C. Allamargot, T. Gesalla, K. Zimmerman, H. Goel, J. M. McLendon, D.-F. Dai, T. Romac, J. Streeter, D. Abhinav, R. O. Pereira, J. Sharafuddin, L.-S. Song, E. D. Abel

This study shows that cardiac-specific overexpression of TFEB leads to pathologic hypertrophy and lethal cardiomyopathy through mitochondrial dysfunction and fibrosis.

bioRxiv · Feb 16, 2021 · PreprintCell Biology