Author

S. Barnes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Guzman· 1 shared A. Hanker· 1 shared A. Mukherji· 1 shared C. Arteaga· 1 shared C. D. D. Cristobal· 1 shared C. D. Morrow· 1 shared C. Johnson· 1 shared C. Rodriguez· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Guzman

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Author profile →

3 papers

Recent·Most cited

ZNF397 Loss Triggers TET2-driven Epigenetic Rewiring, Lineage Plasticity, and AR-targeted Therapy Resistance in AR-dependent Cancers

P. Mu, Y. Xu, Z. Wang, M. Sjostrom, S. Deng, C. Wang, N. Jonhson, J. Gonzalez, X. Li, L. Metang, C. Rodriguez, A. Mukherji, G. Wainwright, X. Yu, Y. Yang, , T. Wang

This study shows ZNF397 loss leads to TET2-driven epigenetic changes, lineage plasticity, and resistance to AR-targeted therapies in AR-dependent cancers, with TET2 inhibitors reversing resistance.

Cancer Discovery · Apr 8, 2024Peer-reviewedCancer Biology

Nutritional combinatorial impact on the gut microbiota and plasma short-chain fatty acids levels in the prevention of mammary cancer in Her2/neu estrogen receptor-negative transgenic mice

M. Sharma, I. Arora, M. L. Stoll, Y. Li, C. D. Morrow, S. Barnes, T. F. Berryhill, S. Li, T. O. Tollefsbol

This study evaluates how broccoli sprouts and green tea polyphenols, alone or combined, affect gut microbiota and short-chain fatty acids in mice to prevent estrogen receptor-negative mammary cancer.

PLOS ONE · Dec 31, 2020Peer-reviewedMicrobiology

Modeling IKZF1 lesions in B-ALL reveals distinct chemosensitivity patterns and potential therapeutic vulnerabilities

J. H. Rogers, R. Gupta, J. M. Reyes, M. Gundry, G. Medrano, A. Guzman, T. Song, C. Johnson, S. Barnes, C. D. D. Cristobal, L. Brunetti, M. A. Goodell, R. E. Rau

This study models IKZF1 lesions in B-ALL to show distinct chemosensitivity patterns and therapeutic vulnerabilities, identifying cytarabine sensitivity and JAK/STAT upregulation as potential targets.

bioRxiv · May 26, 2020Cancer Biology