Author

P. Jia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:proteomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. Barral· 1 shared A. Soussi-Gounni· 1 shared C. Du· 1 shared C. Huang· 1 shared C. I. de Oliveira· 1 shared C. Xu· 1 shared G. Gupta· 1 shared J. Dong· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Barral

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Author profile →

3 papers

Recent·Most cited

PSTPIP2 ameliorates aristolochic acid nephropathy by suppressing interleukin-19-mediated neutrophil extracellular trap formation

C. Du, J. Dong, C. Xu, P. Jia, N. Cai, Q. Wang, Z. Zhang, L. Jiang, W. Jiang, R. Feng, J. Li, C. Huang, T. Ma

This study shows PSTPIP2 reduces kidney damage in AAN by suppressing IL-19-mediated NET formation and inflammation.

bioRxiv · Jul 24, 2023Pharmacology & Toxicology

Ring-finger protein 34 facilitates nervous necrosis virus evading antiviral innate immunity by targeting TBK1 and IRF3 for ubiquitination and degradation

W. Zhang, L. Chen, L. Yao, P. Jia, Y. Xiang, M. Yi, K. Jia

This paper identifies RNF34 as a viral E3 ubiquitin ligase that suppresses antiviral innate immunity by degrading TBK1 and IRF3, facilitating NNV replication.

bioRxiv · Dec 5, 2022 · PreprintImmunology

The Long Pentraxin 3 (PTX3) Suppresses Immunity to Cutaneous Leishmaniasis by Negatively Regulating Th17 Response

G. Gupta, P. Jia, R. Sharma, R. Zayats, S. M. Viana, L. Shan, Z. Mou, V. S. Boaventura, A. Barral, T. T. Murooka, A. Soussi-Gounni, C. I. de Oliveira, J. E. Uzonna

This study shows PTX3 suppresses Th17 responses and increases susceptibility to Leishmania major infection by inhibiting IL-17A production and Th17-specific transcription factors.

bioRxiv · Mar 21, 2019Immunology