Author

J. Ramos

Stanford Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Bioengineering· 1 Biophysics· 1 Cell Biology· 1 Evolutionary Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Approach:statistical model· 4 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Assay:metabolomics· 2 Organism:mouse· 2 Organism:bacteria (other)· 1

Frequent Co-Authors

B. S. Nelson· 3 shared C. A. Lyssiotis· 3 shared C. J. Halbrook· 3 shared H. C. Crawford· 3 shared L. Lin· 3 shared A. Andren· 2 shared A. Myers· 2 shared D. M. Kremer· 2 shared

Activity by Year

2018-2026 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

B. S. Nelson

Mutual co-authors in cluster

Author profile →

9 papers

Recent·Most cited

Leveraging Skin Mutagenesis to Uncover Genetic Cancer Susceptibilities

Audris Chiang, Gayathri Swaminathan, Gaurav Luthria, Marlayna Harris, Haniyah Shareef, Shufeng Li, Vivian J. Hua, Warren Chan, Hyunje Grace Cho, Michelle Jade Lin, Kiana Khalili, J. Ramos, Hanh Do, Mahrukh Abdullah, Irene Bailey, , Kavita Yang Sarin

The paper identifies a novel cancer susceptibility mutation in hfBCC patients by analyzing skin mutagenesis and its association with BCC and prostate cancer.

medRxiv · Oct 6, 2026 · PreprintGenetic & Genomic Medicine

High resolution live cell imaging to define ultrastructural and dynamic features of the halotolerant yeast Debaryomyces hansenii

M. S. C. Xelhuantzi, D. Ghete, A. Milburn, S. Ioannou, P. Mudd, G. Calder, J. Ramos, P. J. O'Toole, P. G. Genever, C. MacDonald

This study uses high-resolution live cell imaging to characterize sub-cellular structures and dynamic processes in the halotolerant yeast Debaryomyces hansenii, providing a template for studying genetically recalcitrant organisms.

bioRxiv · Mar 1, 2024 · PreprintCell Biology

Phylogenomic and genomic analysis reveals unique and shared genetic signatures of Mycobacterium kansasii complex species

E. Machado, S. Vasconcellos, L. Gomes, M. Catanho, J. Ramos, L. de Carvalho, T. Goldenberg, M. Dalcolmo, P. Redner, P. Caldas, C. Campos, M. C. Lourenco, E. Lasunskaia, V. Mussi, L. Spinasse, , P. Suffys

This study analyzes 665 Mycobacterium kansasii complex strains to identify unique and shared genomic signatures, revealing lineage splits, virulence factors, and drug resistance mechanisms.

bioRxiv · Feb 14, 2024Evolutionary Biology

Artificial Antigen Presenting Cells for Detection and Desensitisation of Auto-reactive T cells Associated with Type 1 Diabetes

A. Artzy-Schnirman, E. Abu-Shah, R. Chandrawati, E. Altman, N. Yusuf, S.-T. Wang, J. Ramos, C. Hansel, M. Haus-cohen, R. Dahan, S. Arif, M. L. Dustin, M. Peakman, Y. Reiter, M. M. Stevens

This paper develops artificial antigen presenting cells (aAPCs) to detect and desensitize autoreactive T cells in type 1 diabetes, showing effectiveness in human and mouse models.

bioRxiv · Dec 26, 2020 · PreprintBioengineering

Does retraction after misconduct have an impact on citations? A pre-post study.

C. Candal-Pedreira, A. Ruano-Ravina, E. Fernandez, J. Ramos, I. Campos-Varela, M. Perez-Rios

This study examines whether retraction of articles affects their citations, finding that citations increase post-retraction, except for some cases.

bioRxiv · Aug 11, 2020Scientific Communication & Education

The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2

S. A. Kerk, L. Lin, A. L. Myers, Y. Zhang, J. A. Jimenez, P. Sajjakulnukit, B. S. Nelson, B. Chen, A. Robinson, G. Thurston, S. B. Kemp, N. G. Steele, M. T. Hoffman, H.-J. Wen, D. Long, , C. A. Lyssiotis

This study shows that the pancreatic tumor microenvironment compensates for loss of GOT2 by providing pyruvate, maintaining redox homeostasis and tumor growth in vivo.

bioRxiv · Aug 7, 2020Cancer Biology

GOT1 Inhibition Primes Pancreatic Cancer for Ferroptosis through the Autophagic Release of Labile Iron

D. M. Kremer, B. S. Nelson, L. Lin, E. Yarosz, C. J. Halbrook, S. A. Kerk, P. Sajjakulnukit, A. Myers, G. Thurston, S. Hou, E. S. Carpenter, A. Andren, Z. Nwosu, N. Cusmano, S. Wisner, , C. A. Lyssiotis

This study shows that inhibiting GOT1 in pancreatic cancer primes cells for ferroptosis by promoting autophagy-mediated release of labile iron.

bioRxiv · Feb 28, 2020 · PreprintCancer Biology

Tissue of Origin Dictates GOT1 Dependence and Confers Synthetic Lethality to Radiotherapy

B. S. Nelson, L. Lin, D. M. Kremer, C. M. Sousa, C. Cotta-Ramusino, A. Myers, J. Ramos, T. Gao, I. Kovalenko, K. Wilder-Romans, J. Dresser, M. Davis, H.-J. Lee, Z. C. Nwosu, S. Campit, , C. A. Lyssiotis

The paper shows that pancreatic ductal adenocarcinoma (PDA) is more dependent on GOT1 than colorectal cancer (CRC) and that GOT1 inhibition synergizes with radiotherapy to inhibit PDA growth.

bioRxiv · Jul 25, 2019Cancer Biology

Hydrodynamic and electrophoretic properties of Trastuzumab/HER2 complexes. Experiments and modeling.

J. Ramos, J. F. Vega, V. Cruz, E. Sanchez-Sanchez, J. Cortes, J. Martinez-Salazar

This study combines experiments and modeling to analyze the hydrodynamic and electrophoretic properties of Trastuzumab/HER2 complexes, identifying two distinct complexes and their biophysical characteristics.

bioRxiv · Dec 4, 2018 · PreprintBiophysics