Author

S. Dhawan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Organism:mouse· 4 Research Focus:cancer & tumor microenvironment· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2

Frequent Co-Authors

C. C. Chen· 2 shared J. Ma· 2 shared J. Ning· 2 shared M. Li· 2 shared A. G. J. Skorput· 1 shared A. L. Sarver· 1 shared A. Marta· 1 shared A. Montoya· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersImmunology

C. C. Chen

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

The clock-modulatory activity of Nobiletin suppresses adipogenesis via Wnt signaling

X. Xiong, T. Kiperman, W. Li, S. Dhawan, J. Lee, V. Yechoor, K. Ma

This study shows Nobiletin suppresses adipogenesis by enhancing the circadian clock and activating Wnt signaling, leading to reduced fat mass and body weight in mice.

bioRxiv · Feb 7, 2023 · PreprintCell Biology

Differential CpG methylation at Nnat in the early establishment of beta cell heterogeneity

V. Yu, F. Yong, A. Marta, S. Khadayate, A. Osakwe, S. Bhattacharya, S. S. Varghese, P. Chabosseau, S. M. Tabibi, K. Chen, E. Georgiadou, N. Parveen, M. Suleiman, Z. Stamoulis, L. Marselli, , S. J. Millership

This study identifies differential CpG methylation at the Nnat gene as a mechanism establishing beta cell heterogeneity during development and its potential role in diabetes.

bioRxiv · Feb 4, 2023Cell Biology

Therapeutic activation of virus-specific resident memory T cells within the glioblastoma microenvironment

J. Ning, N. Gavil, S. Wu, S. Wijeyesinghe, E. Weyu, J. Ma, M. Li, F.-N. Grigore, S. Dhawan, A. G. J. Skorput, S. C. Musial, C. C. Chen, D. Masopust, P. C. Rosato

This study shows that virus-specific memory T cells reside in glioblastoma and can be reactivated to improve survival in a mouse model.

bioRxiv · Sep 6, 2021 · PreprintImmunology

PI3Kγ inhibition suppresses microglia/TAM accumulation in glioblastoma microenvironment to promote exceptional temozolomide response

J. Li, M. M. Kaneda, J. Ma, M. Li, K. Patel, T. Koga, A. L. Sarver, F. Furnari, B. Xu, S. Dhawan, J. Ning, H. Zhu, A. Wu, G. You, T. Jiang, , C. C. Chen

The paper identifies PI3Kγ as a driver of microglia/macrophage accumulation in glioblastoma, showing its inhibition enhances temozolomide response in preclinical models.

Proceedings of the National Academy of Sciences · Apr 12, 2021Peer-reviewedCancer Biology