Author

L. Reyes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Evolutionary Biology· 1 Immunology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:host-pathogen & immune response· 4 Assay:bulk RNA-seq· 2 Organism:mouse· 2 Organism:multiple species· 1 Organism:rat· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

A. C. Noguchi· 1 shared A. J. Simpson· 1 shared A. Larsen· 1 shared A. M. Aponte· 1 shared A. M. Mitzey· 1 shared A. M. Weiler· 1 shared A. Rostron· 1 shared A. S. Mirchandani· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 9 mutual co-author links

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1 shared paperPhysiology

A. C. Noguchi

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Author profile →

5 papers

Recent·Most cited

A Narrow Range of Transcript-error Rates Across the Tree of Life

W. Li, S. Baehr, M. Marasco, L. Reyes, D. Brister, C. Pikaard, J.-F. Gout, M. Vermulst, M. Lynch

The paper analyzes transcript-error rates across diverse species, finding a narrow range and suggesting mechanisms for error purging, but the model explains only 20% of the variation.

bioRxiv · May 2, 2023Evolutionary Biology

Vertical transmission of African-lineage Zika virus through the fetal membranes in a rhesus Macaque (Macaca mulatta) model

M. R. Koenig, A. M. Mitzey, X. Zeng, L. Reyes, H. A. Simmons, T. K. Morgan, E. K. Bohm, J. C. Pritchard, J. A. Schmidt, E. Ren, F. L. Jaimes, E. Winston, P. Basu, A. M. Weiler, T. C. Friedrich, , T. G. Golos

This study shows ZIKV can transmit vertically through fetal membranes, not the placenta, in rhesus macaques.

bioRxiv · Mar 13, 2023Pathology

Loss of TMEM65 causes mitochondrial disease mediated by mitochondrial calcium

Y. Zhang, L. Reyes, J. Sun, C. Liu, D. Springer, A. C. Noguchi, A. M. Aponte, J. Munasinghe, R. Covian, E. Murphy, B. Glancy

This study identifies TMEM65's role in mitochondrial calcium regulation, showing its loss causes mitochondrial disease and suggesting therapeutic targeting of mitochondrial calcium as a potential treatment.

bioRxiv · Aug 2, 2022 · PreprintPhysiology

Hypoxia shapes the immune landscape in lung injury promoting inflammation persistence

A. S. Mirchandani, S. J. Jenkins, C. C. Bain, H. Lawson, P. Coelho, F. Murphy, D. Griffith, A. Zhang, M. A. Sanchez-Garcia, L. Reyes, T. Morrison, S. Arienti, P. Sadiku, E. Watts, R. Dickinson, , S. Walmsley

This study shows hypoxia suppresses type I interferon signaling in ALI, leading to monocytopenia and unchecked inflammation, which can be mitigated by CSF1 administration.

bioRxiv · Mar 11, 2022 · PreprintCell Biology

Nano-Emulsion Adjuvant Enhances Cross-presentation by Lipid Body Formation Independent of Glycolysis

M. Suresh, W. Lee, B. Kingstad-Bakke, B. Paulson, A. Larsen, K. Overmyer, C. Marinaik, K. Dull, R. Toy, G. Vogel, K. Mueller, K. Tweed, A. Walsh, J. Russell, K. Saha, , K. Roy

This study shows that the nano-emulsion adjuvant Adjuplex enhances cross-presentation in dendritic cells without requiring glycolysis, relying instead on lipid body formation and ROS.

bioRxiv · May 8, 2020 · PreprintImmunology