Author

S. Eisa-Beygi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

D. B. Cowan· 3 shared H. Chen· 3 shared H. Wu· 3 shared S. Wong· 3 shared A. E. Birsner· 2 shared B. Singh· 2 shared B. Wang· 2 shared B. Zhu· 2 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCell Biology

D. B. Cowan

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3 papers

Recent·Most cited

Interplay Between FoxM1 and Dab2 Promotes Endothelial Cell Responses in Diabetic Wound Healing

S. Bhattacharjee, J. Gao, Y. W. Lu, S. Eisa-Beygi, H. Wu, K. S. Li, A. E. Birsner, S. Wong, Y. Song, J. Y.-J. Shyy, D. B. Cowan, W. Wei, M. Aikawa, J. Shi, H. Chen

This study identifies Dab2 and FoxM1 as key regulators of endothelial dysfunction in diabetes, showing that restoring Dab2 improves angiogenesis and wound healing.

bioRxiv · Feb 7, 2024 · PreprintCell Biology

Epsins oversee smooth muscle cell reprogramming by influencing master regulators KLF4 and OCT4.

B. Wang, K. Cui, B. Zhu, Y. Dong, D. Wang, B. Singh, H. Wu, K. Li, S. Eisa-Beygi, Y. Sun, S. Wong, D. B. Cowan, Y. Chen, M. Du, H. Chen

This study shows Epsins regulate smooth muscle cell reprogramming by modulating OCT4 and KLF4, with implications for coronary artery disease.

bioRxiv · Jan 8, 2024 · PreprintCell Biology

Targeting endothelial FOXO1 protects diabetic β-cells and improves wound healing

H. Wu, K. Cui, B. Zhu, Y. W. Lu, L. Sun, Q. Ma, A. Jian, D. O. Hurtado, X. Gao, Y. Li, S. Bhattacharjee, H. Fu, A. E. Birsner, M. Malovichko, M. Li, , H. Chen

This study shows that targeting endothelial FOXO1 reduces IGF2 levels, improves β-cell function, and enhances wound healing in diabetes.

bioRxiv · Dec 4, 2023 · PreprintCell Biology