Author

E. Mennillo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

I. Rusu· 2 shared J. L. Bain· 2 shared M. G. Kattah· 2 shared M. Naser· 2 shared A. Combes· 1 shared A. Ma· 1 shared A. Marson· 1 shared A. Pisco· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

I. Rusu

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single-cell and spatial multi-omics highlight effects of anti-integrin therapy across cellular compartments in ulcerative colitis

E. Mennillo, Y. J. Kim, G. Lee, I. Rusu, R. Patel, L. C. Dorman, E. Flynn, S. Li, J. L. Bain, C. Andersen, A. Rao, S. Tamaki, J. Tsui, A. Shen, M. L. Lotstein, , M. G. Kattah

This study uses single-cell and spatial multi-omics to show VDZ affects mononuclear phagocyte subsets and highlights gene expression patterns in UC.

bioRxiv · Jan 21, 2023Immunology

Lactational delivery of Triclosan promotes non-alcoholic fatty liver disease in newborn mice

A. Weber, X. Yang, E. Mennillo, J. Ding, J. Watrous, M. Jain, S.-J. Chen, M. Karin, R. Tukey

This study shows that lactational exposure to Triclosan in mice causes fatty liver disease by disrupting metabolic pathways involving PERK-eIF2-ATF4-PPAR.

bioRxiv · May 20, 2022Pharmacology & Toxicology

Microbial signals and lymphotoxin drive TNF-independent death of A20 and ABIN-1 deficient epithelium

I. Rusu, E. Mennillo, J. L. Bain, Z. Li, X. Sun, K. Ly, Y. Y. Rosli, M. Naser, Z. Wang, R. Advincula, P. Achacoso, L. Shao, B. Razani, O. D. Klein, A. Marson, , M. G. Kattah

The paper shows that microbial signals, MyD88, and lymphotoxin contribute to TNF-independent intestinal injury after A20 and Abin-1 deletion in intestinal epithelial cells.

Journal of Clinical Investigation · Mar 1, 2022Peer-reviewedImmunology