R. Lozano-Duran

This study identifies an atypical plant immune receptor, PML5, with a conserved deletion in its signaling domain that mediates cell death and immunity.

This study identifies how the tomato yellow leaf curl virus uses a host-mimicking protein domain to disrupt plant processes and attract insect vectors, facilitating viral spread.

This study shows JAZ8 interacts with geminiviral C2 proteins and limits viral infection in Arabidopsis.

This study shows Ralstonia solanacearum alters root development in Arabidopsis through auxin-dependent and independent mechanisms, including promoting xylem differentiation and root hair formation.

This study shows that phosphorylation of RLP44's C-terminal tail directs its localization to either brassinosteroid or phytosulfokine signalling pathways.

This study shows that viral proteins interact combinatorially, leading to new biological functions beyond their individual roles during infection.

This study identifies additional ORFs in geminiviral genomes that encode small proteins with specific subcellular localizations and virulence functions.

This study identifies host processes targeted by Phytophthora infestans RXLR effectors through protein-protein interaction screens, revealing vesicle trafficking as a major effector-targeted process.

This study identifies DNA polymerases alpha and delta as key mediators of geminivirus replication in plants, with C3 protein selectively recruiting polymerase delta for efficient viral genome synthesis.

This paper identifies a novel protein complex in Arabidopsis that regulates ROS1-mediated active DNA demethylation and targets ROS1 to specific genomic regions.

The paper shows that the TYLCV protein V2 suppresses viral genome methylation in plant Cajal bodies by interacting with AGO4.

This study identifies a pathway connecting plasma membrane and chloroplasts that pathogens exploit to suppress salicylic acid-dependent defenses.

This study identifies IAN9 and IAP1 as negative regulators of basal immunity in Arabidopsis, with potential applications for improving disease resistance in crops.

The paper identifies BAM1 as a positive regulator of RNAi cell-to-cell spread and shows that a viral protein inhibits this process by interacting with BAM1 and BAM2.